OnCo
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TROPION-Breast01: datopotamab deruxtecan versus chemotherapy in pretreated hormone-receptor-positive breast cancer, and why a PFS win did not translate to survival

The TROP2-directed antibody-drug conjugate Dato-DXd delayed progression by about two months compared with chemotherapy, but patients did not live longer, which stalled its approval in breast cancer.

Open-label phase 3 trial of 732 patients with hormone-receptor-positive, HER2-negative metastatic breast cancer after one or two lines of chemotherapy, randomised to datopotamab deruxtecan (Dato-DXd, 6 mg/kg) or investigator's choice chemotherapy (eribulin, vinorelbine, capecitabine or gemcitabine). Dual primary endpoints were PFS by blinded review and overall survival.

PFS was improved (6.9 vs 4.9 months, HR 0.63) with fewer high-grade adverse events, but the final overall survival analysis showed no difference. The trial is a cautionary example that a TROP2 ADC can beat chemotherapy on PFS in an unselected population without changing survival.

Randomised controlled trialHas not changed practice yet732 participants
Authors
Bardia A, Jhaveri K, Im SA, et al.
What it found
  • Median PFS by blinded central review 6.9 vs 4.9 months; HR 0.63 (95% CI 0.52-0.76).
  • Objective response rate 36.4% vs 22.9%.
  • Grade 3 or higher treatment-related adverse events about 21% vs 45%; stomatitis and ocular surface events were the characteristic Dato-DXd toxicities.
  • Final overall survival analysis (2024): no significant difference (HR close to 1.0).
  • TROP2 expression by immunohistochemistry did not select responders.
What it means

For hormone-receptor-positive metastatic breast cancer that has already had chemotherapy, Dato-DXd controls the disease for longer with fewer severe side effects than chemotherapy, but does not help patients live longer, so it is not a standard option here. The result is a reminder that progression-free survival is a surrogate; regulators and clinicians should wait for survival data before adopting an ADC in a setting where later therapies are effective.

Be careful
  • Open-label; PFS assessed by blinded review but subsequent therapy was at physician discretion.
  • Post-progression therapy, including other ADCs, likely diluted any survival effect.
  • The population was chemotherapy-pretreated and heterogeneous; a first-line or biomarker-selected trial might behave differently.
  • Immunohistochemistry for TROP2 was not predictive, leaving no validated way to choose patients.

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