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MajesTEC-1: teclistamab, an off-the-shelf BCMA bispecific antibody, in heavily pretreated myeloma

A ready-made antibody that pulls T cells onto myeloma cells produced responses in 63% of patients after a median of five prior therapies, without the wait for cell manufacturing.

MajesTEC-1 was a phase 1/2 single-arm study of teclistamab, a BCMA x CD3 bispecific T-cell engager given subcutaneously weekly after step-up dosing, in 165 patients with relapsed or refractory multiple myeloma who were triple-class exposed (median five prior lines). The overall response rate was 63%, with complete response or better in 39.4%; median duration of response was 18.4 months and median PFS 11.3 months. Cytokine release syndrome occurred in 72% (almost all grade 1-2) and neurotoxicity in about 14.5%, but infections were frequent (grade 3-4 in about 45%) and hypogammaglobulinaemia was near universal. Teclistamab received accelerated approval in 2022, the first bispecific for myeloma.

Translational studyChanged practice165 participants
Authors
Moreau P, Garfall AL, van de Donk NWCJ, et al.
What it found
  • 165 triple-class-exposed patients; median 5 prior lines.
  • Overall response 63.0%; complete response or better 39.4%; MRD-negativity in 26.7% of all patients.
  • Median duration of response 18.4 months; median PFS 11.3 months.
  • CRS 72.1% (grade 3 0.6%); neurotoxicity 14.5% (ICANS 3%).
  • Infections 76.4% (grade 3-4 44.8%); 5 COVID-19 deaths; hypogammaglobulinaemia common.
What it means

Teclistamab showed that an off-the-shelf bispecific can approach CAR-T-like response rates in late myeloma, giving patients who cannot wait for or access cell therapy a real option. It also exposed the price: prolonged T-cell engagement causes profound immunosuppression, so infection prophylaxis and immunoglobulin replacement are now routine. Less frequent dosing after response is being adopted to reduce this burden.

Be careful
  • Single-arm study; MajesTEC-3 later provided randomised evidence in earlier lines.
  • Continuous weekly dosing until progression in the original protocol; optimal duration and schedule still being defined.
  • Infection-related deaths were substantial; many patients were treated during the COVID-19 pandemic.
  • Responses are shorter than with cilta-cel, and BCMA-directed sequencing (CAR-T then bispecific or vice versa) reduces subsequent efficacy.

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