OnCo
key papersKey paper

AlphaFold 2: predicting protein structures to near-experimental accuracy

DeepMind's neural network predicted protein structures at CASP14 with a median backbone error of under 1 angstrom, comparable to experimental methods, and the team released predicted structures for essentially every human protein within a year.

AlphaFold 2 combines multiple-sequence alignments, an attention-based Evoformer network and an equivariant structure module trained end to end on the Protein Data Bank. At the blind CASP14 assessment in 2020 it achieved a median GDT of about 92 and a median backbone RMSD95 of 0.96 angstrom on the hardest targets, versus 2.8 angstrom for the next-best method.

The model provides per-residue confidence estimates (pLDDT), enabling users to distinguish reliable regions from disordered or uncertain ones. The accompanying AlphaFold Protein Structure Database, built with EMBL-EBI, released predicted structures for the human proteome and later for more than 200 million proteins.

For cancer drug discovery, AlphaFold accelerated structure-based design for targets without crystal structures and, with AlphaFold 3 (2024) extending to protein-ligand and protein-nucleic acid complexes, is now a routine part of the target-to-lead pipeline.

MethodsHas not changed practice yet
Authors
Jumper J, Evans R, Pritzel A, et al.
Published
Nature, 2021
What it found
  • CASP14: median backbone RMSD95 of 0.96 angstrom (95% CI 0.85-1.16) vs 2.8 angstrom for the next-best method
  • Median GDT score around 92 across CASP14 targets, the first time a computational method reached experimental-grade accuracy
  • Per-residue confidence (pLDDT) reliably flags disordered and low-confidence regions
  • Predicted structures for the entire human proteome released in 2021; over 200 million proteins by 2022
What it means

The shape of nearly every protein is now available to any researcher in seconds instead of years, which shortens the path from a cancer target to a designed molecule. It does not by itself produce drugs: binding pockets, dynamics and cellular context still need experiment.

Be careful
  • Predicts single static conformations; many drug targets (kinases, GPCRs, KRAS) move between states
  • Accuracy is lower for proteins without evolutionary homologues, disordered regions and multi-protein complexes
  • Does not predict effects of point mutations or ligand binding (AlphaFold 3 and other tools partly address this)
  • The 2021 model was released with a non-commercial licence for weights, later loosened

Connected

10top